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Almost tears as my prof Vogl addressed conference

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It's his conference,  leading Germany unis. I sit in front row, I'm a guest of vogl, his miracle as much as anyone else's.

I breathe and thank god for my life, I talked to everyone I could speakers, radiologists, suppliers and waiters.  I look at the pictures on the big screen trying to guess what the speakers were saying in German. I must learn German.

I prayed to god to help me understand why he leads me here and there and why my challenge.

I got something somehow from every speaker and an invite to meet a top lung cancer radiologist. I asked about other young interventions radiologists who want to work with me. ALL ANSWERED ONLY VOGL. But i still crave to meet fellow cancer research scientists, there is so much much more i could learn if i had a team behind me.

My old brave friend Winter Marie from csn the cancer survivors network, recently at rest and now at peace condemned vogl, as many others as well. It seems whatever I suggest or praise gets rubbished and attacked. Just like my love affair with gcmaf going on past 4 years. I am at home with innovators, I seem to attract and retain them.

So I have my vogl on a higher pedestal than even before, my respect not based on my miracle alone but the obvious respect of the most dedicated German radiologists I was surrounded by. They wished me much success, I glowed within, I wonder of my bliss is transparent. My bliss was more cognitive the last few days.

A new friend sells software that analyzes ct and mri liver mets and does surgical plans. Artificial intelligence my old favorite in my computing science degree.

I'm returning the hire car to save 50 euro
as its Sunday tomorrow and I don't need a car. This proved difficult, so I parked and walk the streets of frankfurt in the less glamorous area around main station and the river.


http://scienceblog.cancerresearchuk.org/2008/12/03/cancer-cured-for-good-gc-maf-and-the-miracle-cure/

Some companies are selling Gc-MAF for use by cancer patients. This treatment is not approved or licensed in the UK for treating cancer or any 0ther disease. Given that there is no solid scientific evidence to show that the treatment is safe or effective, we would not recommend that cancer patients use it. [Updated KA 25/07/14] 

SO THE CHARITY ABOVE HAS POSSIBLY KILLED ONE OR MILLION, LIKE THE EVIDENCED BASED ONCOLOGISTS.  SO MANY DESPERATE PAIN RIDDEN CANCER PATIENTS TAKE THERE ADVICE. I NO LONGER NO WHATS RIGHT OR WRONG. I THANK GOD FOR MY LIFE AND I FREELY SHARE MY IRRATIONAL HOPE HERE. I THOUGHT OF MY FAMILY ASLEEP IN SYDNEY WHILE SURROUNDED BY SLEEPY RADIOLOGISTS AFTER LUNCH. 

I ENJOYED THE COMPANY OF 3 STUNNING YOUNG GIRLS AT THE CONFERENCE DRINKS, ALL RADIOLOGISTS FROM BERLIN. I ENTERTAINED THEM WITH AMAZING TALES AND WENT HOME ALONE TO MY DINGY HOTEL, BUT COMPARED TO THE HOMELESS I WALK PAST, WELL MY ROOM IS MY PALACE.

I KNOW MY WIFE LOVES ME, I AM SO BLESSED IN EVERY WAY.

I DO CHERISH THESE EXPERIENCES.


SO I AM THE WORLDS FIRST INTERVENTIONAL METABOLIC IMMUNOTHERAPIST WITH AN INTEREST IN CANCER GENETIC MUTATIONS

AFTER ALL THE SCIENCE. MAYBE THE SIMPLE PRAY I LOVES YOU ENOUGH TO SAVE ME. WHO KNOW.  I SAY MY PRAYERS AND READ MY SCIENCE.

http://www.ncbi.nlm.nih.gov/pubmed/22936024

Abstract

Dendritic cells (DCs) and monocyte-derived macrophages (MΦs) are key components of intestinal immunity. However, the lack of surface markers differentiating MΦs from DCs has hampered understanding of their respective functions. Here, we demonstrate that, using CD64 expression, MΦs can be distinguished from DCs in the intestine of both mice and humans. On that basis, we revisit the phenotype of intestinal DCs in the absence of contaminating MΦs and we delineate a developmental pathway in the healthy intestine that leads from newly extravasated Ly-6C(hi) monocytes to intestinal MΦs. We determine how inflammation impacts this pathway and show that T cell-mediated colitis is associated with massive recruitment of monocytes to the intestine and the mesenteric lymph node (MLN). There, these monocytes differentiate into inflammatory MΦs endowed with phagocytic activity and the ability to produce inducible nitric oxide synthase. In the MLNs, inflammatory MΦs are located in the T-cell zone and trigger the induction of proinflammatory T cells. Finally, T cell-mediated colitis develops irrespective of intestinal DC migration, an unexpected finding supporting an important role for MLN-resident inflammatory MΦs in the etiology of T cell-mediated colitis.


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