IT turns out the fever was an infected port, i had the same experience a few days later without edta. we always look for more complex answers, sometimes the obvious is the way to go, or assume. karin the nurse was spot on, what an asset to hallwang. of course kopic handled my episode perfectly, we were all a cool as cucumbers emotionally and medically, alas i was a hot potatoe in a manner of speaking.
ORIGINAL
I was originally just getting the p53 suppressor therapy today, but its being sent tuesday, so should arrive wednesday. I will then leave that afternoon and goto duderstadt for gamma delta cell therapy.
So I asked if I could have EDTA today as I try and get this once per week, Now I will put that request on hold Today about 2 hours into a 4 four etda drip I started to feel really tired.
When the nurse checked my temp it was 37.1, my normal temp is always 36.6. somethings wrong. my temp climbed to 38.4, during this rise I started to shake, I had a couple of blnkets put on. The nurse called Dr Kopic, some IV paracetamol and in about about 15 minutes I started to feel better.
The EDTA drip was stopped, other basic infusions were given. I was going to have the port needle out, but advised to leave it in over night.
The nurse is very experienced, she has never seen this reaction and does not believe its edta. I think it was and that something in my bio chemistry contributed to this. The way my reation was handled was excellent by karin and asir kopic.
The interation between etda and ketogenic diet warrants further investigation.
http://www.livestrong.com/article/24863-edta-chelation-side-effects/
the side effects of edta and immune suppression, well to be honest I forgot about this one. I will put my edta chelation plans on hold for the time being.
Is it something to do with the ketogenic diet, my blood sugars were normal. mybp was slightly elevated. our biology is amazing and complex. I will continue reseaching etda and ketogenic and also hyperthermia.
http://www.livestrong.com/article/545200-the-fall-of-fitness/
Uncle Rhabdo represents a character in the CrossFit community and is short for rhabdomyolysis, a kidney condition most commonly induced by excessive exercise, according to Heather Gillespie, a sports medicine physician from UCLA. The potentially life-threatening state, which can also be caused by underlying genetics, occurs when muscle breaks down and myoglobin, the biproduct of muscle fibers, is released into the blood stream, essentially clogging up the kidneys and poisoning them.
“If you’re dehydrated, which sort of goes along with rhabdo, you can’t clear these toxins, the kidney can’t filter the byproduct,” Gillespie says. It can lead to kidney failure and electrolyte imbalances that can ultimately affect your heart.
Uncle Rhabdo was originally invented to shed light on “the inappropriate use of intensity,” according to CrossFit’s Training Guide
Read more: http://www.livestrong.com/article/545200-the-fall-of-fitness/#ixzz2OxZJE19o
the implications for crossfit, which is similar to what my personal trainers are recommending. but I have been doing a mild version of this, the balance between healing and vigorous exercise a difficult juggle. For me to check my heart rate up for 30 minutes after breakfast and then for 30 minutes weight training most nights is enough.
http://intelegen.com/nutrients/chelation.htm
Kindness and Frackelton reported several years ago that EDTA has a profound effect on thrombin-induced platelet aggregation. Platelet aggregation is an essential step in the formation of a blood clot. Intra-arterial blood clots are a major cause of heart attacks and strokes, especially when the vessel has been narrowed by atherosclerotic plaque. Kindness and Frackelton demonstrated that this physiological anti-clotting property of EDTA is superior to that of aspirin, without the dangers inherent in aspirin therapy.7
I believe that one of the least-touted but most significant mechanisms of EDTA may be that of its ability to "resuscitate mitochondria." Mitochondria are the "power plants" of every cell in the body. (Fig. 2) It is within the mitochondria that the process of oxidative phosphorylation takes place, which generates energy-producing ATP without which life could not exist. Loss of mitochondrial function has long been considered to be one of the primary causes of the aging process.8,9,10 Recently, the role of impaired mitochondrial function in the pathogenesis of many diseases has been increasingly recognized.11,12,13 EDTA was recognized as early as the 1950's to have the ability to stabilize mitochondria.14 Hunter and colleagues suggested that this effect of EDTA was likely to be due to its combining with the mitochondrial membrane rather than its chelating with metal ions.15 Gallagher's studies tended to confirm their belief.14
.....
Other potential adverse effects include hypocalcemia (excessively low blood levels of calcium) due to EDTA's binding excessively with calcium in the blood; hypoglycemia (low blood sugar), believed to be due to accompanying hypocalcemia; and phlebitis (inflammation of the vein) usually due to improperly prepared solutions. Rarely reported side effects include chills and fever following infusion, exacerbation of congestive heart failure due to fluid overload, fatigue (usually due to hypoglycemia or hypocalcemia), seizures, arrythmias, or rash. Although these have been reported occasionally by others, I have never observed any of these rare side effects despite having administered thousands of IV EDTA treatments.
http://www.auntminnie.com/index.aspx?sec=ser&sub=def&pag=dis&ItemID=1570
ORIGINAL
I was originally just getting the p53 suppressor therapy today, but its being sent tuesday, so should arrive wednesday. I will then leave that afternoon and goto duderstadt for gamma delta cell therapy.
So I asked if I could have EDTA today as I try and get this once per week, Now I will put that request on hold Today about 2 hours into a 4 four etda drip I started to feel really tired.
When the nurse checked my temp it was 37.1, my normal temp is always 36.6. somethings wrong. my temp climbed to 38.4, during this rise I started to shake, I had a couple of blnkets put on. The nurse called Dr Kopic, some IV paracetamol and in about about 15 minutes I started to feel better.
The EDTA drip was stopped, other basic infusions were given. I was going to have the port needle out, but advised to leave it in over night.
The nurse is very experienced, she has never seen this reaction and does not believe its edta. I think it was and that something in my bio chemistry contributed to this. The way my reation was handled was excellent by karin and asir kopic.
The interation between etda and ketogenic diet warrants further investigation.
http://www.livestrong.com/article/24863-edta-chelation-side-effects/
the side effects of edta and immune suppression, well to be honest I forgot about this one. I will put my edta chelation plans on hold for the time being.
Is it something to do with the ketogenic diet, my blood sugars were normal. mybp was slightly elevated. our biology is amazing and complex. I will continue reseaching etda and ketogenic and also hyperthermia.
http://www.livestrong.com/article/545200-the-fall-of-fitness/
Uncle Rhabdo represents a character in the CrossFit community and is short for rhabdomyolysis, a kidney condition most commonly induced by excessive exercise, according to Heather Gillespie, a sports medicine physician from UCLA. The potentially life-threatening state, which can also be caused by underlying genetics, occurs when muscle breaks down and myoglobin, the biproduct of muscle fibers, is released into the blood stream, essentially clogging up the kidneys and poisoning them.
“If you’re dehydrated, which sort of goes along with rhabdo, you can’t clear these toxins, the kidney can’t filter the byproduct,” Gillespie says. It can lead to kidney failure and electrolyte imbalances that can ultimately affect your heart.
Uncle Rhabdo was originally invented to shed light on “the inappropriate use of intensity,” according to CrossFit’s Training Guide
Read more: http://www.livestrong.com/article/545200-the-fall-of-fitness/#ixzz2OxZJE19o
the implications for crossfit, which is similar to what my personal trainers are recommending. but I have been doing a mild version of this, the balance between healing and vigorous exercise a difficult juggle. For me to check my heart rate up for 30 minutes after breakfast and then for 30 minutes weight training most nights is enough.
http://intelegen.com/nutrients/chelation.htm
Kindness and Frackelton reported several years ago that EDTA has a profound effect on thrombin-induced platelet aggregation. Platelet aggregation is an essential step in the formation of a blood clot. Intra-arterial blood clots are a major cause of heart attacks and strokes, especially when the vessel has been narrowed by atherosclerotic plaque. Kindness and Frackelton demonstrated that this physiological anti-clotting property of EDTA is superior to that of aspirin, without the dangers inherent in aspirin therapy.7
I believe that one of the least-touted but most significant mechanisms of EDTA may be that of its ability to "resuscitate mitochondria." Mitochondria are the "power plants" of every cell in the body. (Fig. 2) It is within the mitochondria that the process of oxidative phosphorylation takes place, which generates energy-producing ATP without which life could not exist. Loss of mitochondrial function has long been considered to be one of the primary causes of the aging process.8,9,10 Recently, the role of impaired mitochondrial function in the pathogenesis of many diseases has been increasingly recognized.11,12,13 EDTA was recognized as early as the 1950's to have the ability to stabilize mitochondria.14 Hunter and colleagues suggested that this effect of EDTA was likely to be due to its combining with the mitochondrial membrane rather than its chelating with metal ions.15 Gallagher's studies tended to confirm their belief.14
.....
Other potential adverse effects include hypocalcemia (excessively low blood levels of calcium) due to EDTA's binding excessively with calcium in the blood; hypoglycemia (low blood sugar), believed to be due to accompanying hypocalcemia; and phlebitis (inflammation of the vein) usually due to improperly prepared solutions. Rarely reported side effects include chills and fever following infusion, exacerbation of congestive heart failure due to fluid overload, fatigue (usually due to hypoglycemia or hypocalcemia), seizures, arrythmias, or rash. Although these have been reported occasionally by others, I have never observed any of these rare side effects despite having administered thousands of IV EDTA treatments.
Challenging results from naturopathic tests
your Metabolism aktivity test is back and showed a dysbiosis of your intestinal system with an inflammed situation and also a mycosisincreasement in the intestinal system. The Immunsystem in generell shows a inflammatory situation. Your Liver and Kidney detox funktions are reduced. these are the conclusions of cindy my naturopath from a recent test, i wonder how relevant these results are to my edta reaction. I will ask Kopic to assess my liver and kidney function. EDTA ability to stimulate mitochondria and its anti clotting. Possibly it could be the celebrex I am taking. this is another area for me to search.
p53 suppressor therapy delayed still researching
http://www.powershow.com/view/3c018f-MGNmO/Gene_therapy_Gene_therapy_to_correct_a_genetic_defect_b_powerpoint_ppt_presentationhttp://www.auntminnie.com/index.aspx?sec=ser&sub=def&pag=dis&ItemID=1570
Multiple dose therapy confers "a highly significant survival advantage," according to Dr. Buller. He noted that the survival rate compares favorably to a 15-month survival of newly recurrent ovarian cancer treated with Taxol.
"This combination treatment shows a synergy between chemotherapy and gene therapy," Dr. Buller told Reuters Health. Forty-four percent of patients who completed three cycles of therapy showed a greater than 50% decrease in the surrogate CA125 tumor marker, which Dr. Buller said "is virtually unheard of in heavily pretreated patients."
"Ovarian cancer is well-suited for gene therapy treatment, because it presents as advanced, metastatic disease, which generally is confined within the peritoneal cavity," Dr. Buller said. He and an international group of investigators recently began a large randomized phase III trial of p53 gene therapy in the treatment of primary ovarian cancer. Patients will receive carboplatin and Taxol, or carboplatin, Taxol and p53 gene therapy.
http://link.springer.com/article/10.1007%2FBF00666204
http://jnci.oxfordjournals.org/content/88/20/1442.full.pdf bingo gold, great article explaining why my choosen therapies might save my life. And indirectly why hallwang is the best place on the planet for me. Hallwang is not doing HT with gene therapy, luckily I am heading to duderstadt and will have local HT.
I have to check if a conflict exists between gamma delta and the p53 gene therapy I am having.
A phase I, retrovirusmediated wild-type p53 gene therapy of lung cancer has recently been reported {288). No clinically significant vector-related
toxicity was noted. Whereas local tumor regression was reported
in three of nine lung cancer patients who had previously failed
conventional therapy, the efficacy of p53 gene therapy will be
determined in studies designed to address this issue. p53 gene
therapy can be coupled with either cancer chemotherapeutic
agents or ionizing radiation. The mechanism of cell death
mediated by p53 was shown in some studies to occur via the
apoptotic pathway [reviewed in (232234-236)]. Da Costa et al.
(237) have devised a novel strategy of gene therapy in which the
mutant p53 in tumor cells binds to exogenouslyintroduced gene
products, resulting in transcriptional activation of a toxic gene.
The results of these successful laboratory studies using retroviral and adenoviral p53 expression vectors have led to the approval of phase I protocols in humans (Table 3). Whereas gene
therapy is conceptually simple and the laboratory results are encouraging, significant obstacles, e.g., incomplete targeting of the
tumor cell population, may limit the success of the current
human trials [reviewed in (238-240)]. Nevertheless, improvements in the biotechnology of gene therapy can be anticipated,
and the strategy of combining p53 gene therapy with other
therapeutic modalities may be more efficacious.
http://jnci.oxfordjournals.org/content/88/20/1442.full.pdf bingo gold, great article explaining why my choosen therapies might save my life. And indirectly why hallwang is the best place on the planet for me. Hallwang is not doing HT with gene therapy, luckily I am heading to duderstadt and will have local HT.
I have to check if a conflict exists between gamma delta and the p53 gene therapy I am having.
A phase I, retrovirusmediated wild-type p53 gene therapy of lung cancer has recently been reported {288). No clinically significant vector-related
toxicity was noted. Whereas local tumor regression was reported
in three of nine lung cancer patients who had previously failed
conventional therapy, the efficacy of p53 gene therapy will be
determined in studies designed to address this issue. p53 gene
therapy can be coupled with either cancer chemotherapeutic
agents or ionizing radiation. The mechanism of cell death
mediated by p53 was shown in some studies to occur via the
apoptotic pathway [reviewed in (232234-236)]. Da Costa et al.
(237) have devised a novel strategy of gene therapy in which the
mutant p53 in tumor cells binds to exogenouslyintroduced gene
products, resulting in transcriptional activation of a toxic gene.
The results of these successful laboratory studies using retroviral and adenoviral p53 expression vectors have led to the approval of phase I protocols in humans (Table 3). Whereas gene
therapy is conceptually simple and the laboratory results are encouraging, significant obstacles, e.g., incomplete targeting of the
tumor cell population, may limit the success of the current
human trials [reviewed in (238-240)]. Nevertheless, improvements in the biotechnology of gene therapy can be anticipated,
and the strategy of combining p53 gene therapy with other
therapeutic modalities may be more efficacious.